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Diet & Life-style may strain your body's
proper control of blood sugar levels and often leads to
insulin insufficiency and erratic secretion of insulin -
precursors to diabetes
Research shows that when the body has optimal nutrition
available that is specific to pancreatic and Immune
System support, plus the electromagnetic directives to
act on that nutritional input, it can manage insulin
production, output and use more efficiently, and can
rebuild pancreatic tissues and beta cell formations
damaged by a diet disproportionately high in sugars.
Sometimes we tend to eat too many simple carbohydrates
(sugar, ice cream, crackers and cookies etc.) which
tends to put a strain on the body's ongoing ability to
deal with such overly refined foods. Our body, although
miraculous, can wear out when asked to live off of a
long- term compromised, overly processed diet.
And when our diets have become compromised, we usually
have various problems including suffering from the
"sugar blues," those deep pockets of low energy reserves
that we may feel after a meal (insufficiently balanced
with protein and long chain carbohydrates) when we tend
to get sleepy and look for the nearest couch.
Unfortunately, such strain over time can lead to serious
problems of Insulin insufficiency and erratic secretion,
pancreatic disease, hormonal imbalance and Insulin
receptor abnormalities, all of which may culminate into
diabetes mellitus, unless the body can be rebalanced and
restored through the proper diet.
Whether
hypo- or hyperglycemic (pre-diabetic), or diabetic, as
well as to help the body maintain appropriate Insulin
sensitivity. The
inclusion of a safe
chromium source in one's diet has been proven to assist the body in controlling
Insulin production and use, are particular and specific
for the nutritional support of diabetes mellitus and
diabetic ulcers, pancreatic disease and other related
glandular disorders. Proper nutrition helps rebuild and heal pancreatic tissues, ß-cell
formations, control Insulin management, output and use
while subsequently decreasing the amount of sugar in the
urine.
Indeed, diabetes mellitus is the most common of any
serious metabolic disease in humans, and perhaps more
than any other disease, is closely associated with diet.
Moreover, diabetic populations are significantly higher
where western lifestyle and diet habits dominate rather
than those cultures consuming a more "primitive" diet.
Four percent of America now suffers with diabetes; 90%
of these are Type II and the remaining 10% are Type I.
The prevalence of diabetes is rising. It's now the 7th
major cause of death in the U.S. and it's thought that
this will double every 10-15 years, with an estimated
6-10% increase per year.
Diabetes mellitus is a chronic disorder of carbohydrate,
fat and protein metabolism, generally characterized by
fasting elevations of blood glucose levels and erratic
Insulin management production, with subsequent increased
chances for developing atherosclerosis, kidney disease
and loss of nerve function.
Insulin is a peptide hormone secreted by the ß-cells of
the islets of Langerhans of the pancreas in response to
an elevation in blood glucose or other secretagogues. It
plays a crucial role in glucose homeostasis by
regulating the uptake and metabolism of glucose by
peripheral tissues and the production and storage of
glucose by the liver. Insulin also regulates the
metabolism of lipids and proteins, the synthesis of
nucleic acids and the expression of certain genes. In
some cells, and perhaps in fetal life, Insulin also has
a less well-defined role as a growth factor.
Type I
is an Insulin-dependent diabetes
(IDDM), now known to be a T-cell mediated autoimmune
disease specifically targeting the pancreatic beta
cells, a deficiency strongly correlated to a hereditary
predisposition to injury or destruction of pancreatic
ß-cells which produce and secrete Insulin, and the lack
of the respective tissue regenerative power of those
cells. The ß-cell insufficiency and destruction is
generally caused by chemical-pH imbalances and viral or
antibody damage such as that caused by inflammatory
cytokines, particularly those produced by Th1-type
lymphocytes, which are hypothesized to play a major role
in the pathogenesis of all autoimmune diseases,
including diabetes of this type, susceptible to
individuals at an early age - usually childhood onset.
Type II
is a non-Insulin dependent
diabetes (NIDDM), being a disorder of glucose
homeostasis characterized by hyperglycemia, peripheral
Insulin resistance, impaired hepatic glucose metabolism,
and diminished glucose-dependent secretion of Insulin
from pancreatic ß-cells. This latter defect may lie in
the glucose signaling pathway in ß-cells involving
metabolically regulated Potassium channels which are the
targets of sulphonylurea drugs commonly used in the
treatment of NIDDM. Type II is characterized by Insulin
insensitivity evidenced by typically high levels of
circulating Insulin and the reversibility of blood sugar
elevation by dietary changes and/or weight loss
sufficient to restore Insulin sensitivity. Low GTF
chromium levels are a major determinant of Insulin
insensitivity, and obesity is yet another significant
factor; onset is generally diet related and usually
occurs later in life.
Simple herbs are believed to lower blood
glucose levels, and quintessential Branched Chain Amino
Acids (BCAAs), which act to increase Insulin sensitivity
within the circulatory system, increase appropriate
fatty acid synthesis and may be indirectly involved with
interleukin and interferon secretion by lymph cells.
Indeed, recently BCAAs and other large neutral Amino
Acids have been found to be beneficial when elevated
within the blood plasma of diabetic individuals. BCAAs
may also help to protect against hypertension and
cardiovascular problems now known to be associated with
diabetic onset.
The Prickly Pear (Opuntia steptacantha, commonly known
as "Nopal" in Mexico)
In a study reported in Diabetes Care and later in the
Science News, Vol. 133, No.4, January 1988, this
particular desert cactus was shown to lower blood
glucose and Insulin levels in diabetics, the authors
believing that the Prickly Pear treatment may improve
the ability of Insulin to efficiently stimulate the
movement of glucose from the blood into body cells.
L-Alanine: Most highly concentrated amino acid
released by muscle groups, functioning as a major energy
source in the body; an important participant and
regulator in glucose metabolism.
BCAAs: L-Leucine, L-Valine & L-Isoleucine:
Specific branched chain amino acids which act to
increase insulin sensitivity within the circulatory
system and increase appropriate fatty acid synthesis;
may also help to protect against hypertension and
cardiovascular problems now known to be associated with
the onset of diabetes.
Radix Trichosanthis: Helps the body naturally use
fatty substances, providing provisions for energy which
puts less pressure on other metabolic pathways and
stores (ie, insulin-glycogen-glucose).
Fenugreek Seeds (Trigonella Foenum-Graecum):
Contains an alkoloid which demonstrates hypoglycemic
activity, and found in studies to exert
cholesterol-lowering and blood sugar lowering effects.
Prickly Pear (Opuntia spp.): Shown to lower blood
glucose and insulin levels in diabetics; believed to aid
the body's ability to effectively monitor insulin levels
by moving glucose from blood into body cells.
Chromium, proven to assist the body in
controlling insulin production and use.
Reishi Mushroom: Contains phytomedicinals
demonstrated to lower blood glucose levels in diabetics.
Vitamins, Micro-Nutritionals & More: Further
supports the rebalance and maintenance of the body's
metabolism of fats, carbohydrates and protein, aiding in
the proper management of blood glucose levels.
Diabin+ (our
featured product) is a safe and effective formulation which is
further strengthened with the addition of Vaxa'ss
TriCardia+ (containing 32-Free Form Amino Acids) and
a diet rich in natural
Probiotics easy and inexpensive to do right in your
own home.
New published research indicates that there is
now good and compelling reason to supply the body with
milk-based proteins, especially those which are
predominantly Casein in nature like those contained
within the Systemex. In this French study originating
from the Sainte-Marguerite Hospital in Marseille, a diet
rich in Casein appears to actually protect subjects
(non-obese mice who have a genetic predisposition for
developing diabetes: NOD mice) from developing diabetes
and then passing it on to their young. Specifically,
Casein fed NOD female mice were protected against
spontaneous diabetes and male NOD mice against acute
Cyclosphosphamide or Cy-induced diabetes while also
lessening the severity of insulitis.
Moreover, Casein has been found to exhibit the highest
ratio of Total Essential Amino Acids to total Nitrogen
of all foods and proteins reported by the FAO/WHO Expert
Group.
The specific Amino Acid configuration of Casein just
mentioned appears to effectively compete against the
inflammatory cytokines response produced by the
lymphocytes of the Immune System mentioned earlier, and
may even reduce or circumvent the possibility of such an
aberrant response, protecting beta cell integrity within
the pancreas and the subsequent production of Insulin
from amino acids derived from its structure. No other
changes in the Immune System could account for these
results. Such may also allow or ensure ß-cell "rest," a
treatment strategy now of medical preference.
Interestingly, egg-based (albumin) proteins
and other hydrolyzed proteins did not
demonstrate this "protective" effect;
Subjects fed albumin based proteins
developed insulitis in 10 weeks. This is yet
another strong reason for those who have a
family history of diabetes, especially
pregnant or lactating women and children, to
supplement with Vaxa's
TriCardia+. Because of the autoimmune
nature of diabetes, supplementation with our
pre-probiotics (kombucha
tea, ginger beer, water kefir and
tibicos) offer appropriate dispersal of
toxins while supporting and boosting the
immune system.
pre-diabetic
or diabetic
kefir -
Caspian Sea Yogurt - Viili
These milk cultures are easily made at home,
require no cooking, very little preparation,
and children of all ages love it. And
there're healthy! They are full of active
(not dead) probiotics, enzymes, nutrients
and Casein. A French study originating from
the Sainte-Marguerite Hospital in Marseille,
found a diet rich in Casein appears to
actually protect subjects (non-obese mice
who have a genetic predisposition for
developing diabetes: NOD mice) from
developing diabetes and then passing it on
to their young. Specifically, Casein fed NOD
female mice were protected against
spontaneous diabetes and male NOD mice
against acute Cyclosphosphamide or
Cy-induced diabetes while also lessening the
severity of insulitis. Moreover, Casein has
been found to exhibit the highest ratio of
Total Essential Amino Acids to total
Nitrogen of all foods and proteins reported
by the FAO/WHO Expert Group. |
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presented by
www.HappyHerbalist.com
Ed Kasper L.Ac, Acupuncturist & Herbalist
417 Laurent St. Santa Cruz, CA 95060
email:
eddy@happyherbalist.com
to order
Diabetin click here
Our liquid extracts are the most potent form of a
botanical elixir.
The Happy Herbalist’s Pharmacy features
fresh-made Remedies Designed Exclusively For You
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us design a formula exclusively for you click here
References:
Arfeen S; Goodship TH; Kirkwood A; Channon S; Ward MK,
"1% amino acid peritoneal dialysate: single-cycle study in diabetic
patients with end-stage renal disease," Department of Internal Medicine,
University of Missouri Health Sciences Center, Am J Kidney Dis 1994
Jan;23(1):86-90.
Arden SD; Roep BO; Neophytou PI; Usac EF; et al., "Imogen
38: a novel 38-kD islet mitochondrial autoantigen recognized by T cells
from a newly diagnosed type 1 diabetic patient," Department of Clinical
Biochemistry, University of Cambridge, Addenbrooke's Hospital, United
Kingdom, J Clin Invest 1996 Jan 15;97(2):551-61.
Blackwood, A.L., M.D.; Manual of Materia Medica,
Therapeutics and Pharmacology, Second Edition, Chicago, 1922. Birk OS;
Douek DC; Elias D; Takacs K; et al., "A role of Hsp60 in autoimmune
diabetes: analysis in a transgenic model, "Medical Research Council
Clinical Sciences Centre, Royal Postgraduate Medical School, Hammersmith
Hospital, London, United Kingdom, Proc Natl Acad Sci U S A 1996 Feb
6;93(3):1032-7.
Clark, John Henry, "Dictionary of Practical Materia
Media," Ninth Edition, London, 1901. Dewey,W.A., M.D.; Practical
Homeopathic Therapeutics, Third Edition, San Francisco, 1934.
Hancock WW; Polanski M; Zhang J; Blogg N; Weiner HL,
"Suppression of insulitis in non-obese diabetic (NOD) mice by oral
Insulin administration is associated with selective expression of
interleukin-4 and -10, transforming growth factor-beta, and
prostaglandin-E," Department of Pathology, New England Deaconess
Hospital, Boston, Am J Pathol 1995 Nov;147(5):1193-9.
Hermitte L; Atlan-Gepner C; Payan MJ; Mehelleb M;
Vialettes B, "Dietary protection against diabetes in NOD mice: lack of a
major change in the Immune System," Service de Nutrition, Endocrinologie,
Maladies metaboliques, Hospital Sainte-Marguerite, Marseille, France,
Diabete Metab 1995 Oct; 21(4):261-8.
Hopkins BA; Rakes AH; Daniel TE; Zimmerman CA; Croom
WJ Jr., "Effects of intraperitoneal L-leucine, L-isoleucine, L-valine,
and L-arginine on milk fat depression in early lactation cows,"
Department of Animal Science, North Carolina State University, J Dairy
Sci 1994 Apr;77(4):1084-92.
Huges, Richard, M.D., "Principles and Practice of of
Homeopathy," fourth edition, London, 1901. Johnson-Tardieu JM; Walworth
EW; Cornelius JG; Ye X; et al., "Autoimmune diabetes-prone NOD mice
express the Lyt2 alpha (Lyt2.1) and Lyt3 alpha (Lyt3.1) alleles of
CD8,"Department of Pathology & Laboratory Medicine, University of
Florida College of Medicine, Immunogenetics 1996;43(1-2):6-12.
Kendrew, Sir John; "The Encyclopedia of Molecular
Biology," Blackwell Publishers, Oxford, Oxfordshire, England, 1994.
Pieper GM; Jordan M; Adams MB; Roza AM, "Syngeneic
pancreatic islet transplantation reverses endothelial dysfunction in
experimental diabetes," Department of Transplant Surgery, Medical
College of Wisconsin, Diabetes 1995 Sep;44(9):1106-13.
Ruilope LM, "Effects of angiotensin converting enzyme
inhibitors on the progression of diabetic nephropathy." Unidad de
Hipertension, Hospital 12 de Octubre, Madrid, Spain, J Hypertens Suppl
1995 Aug;13(2):S91-3.
Takacs K; Douek DC; Altmann DM, "Exacerbated
autoimmunity associated with a T helper-1 cytokine profile shift in
H-2E-transgenic mice," Clinical Sciences Centre, Royal Postgraduate
Medical School, Hammersmith Hospital, London, UK, Eur J Immunol 1995
Nov; 25 (11):3134-41.
Zipris D; Greiner DL; Malkani S; Whalen B; et
al., "Cytokine gene expression in islets and thyroids of BB rats. IFN-gamma
and IL-12p40 mRNA increase with age in both diabetic and Insulin-treated
nondiabetic BB rats," Department of Medicine, University of
Massachusetts Medical Center, J Immunol 1996 Feb 1;156(3):1315-21.
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